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This means Ligandrol works in a similar way to testosterone and anabolic steroids, although SARMs typically have fewer side effectsand longer duration of action than testosterone, anabolic steroids and synthetic testosterone. "These new medicines are used to treat many different conditions, yet their main mechanism of action is the regulation of gene expression," says Michael R. Wohlman, Ph.D., an associate research scientist with the Department of Pharmacology at the University of Iowa College of Pharmacy. The molecules, which contain five non-receptor tyrosine kinase domains and six non-receptor tyrosine phosphatase domains, interact via a protein called the SRT1 kinase receptor tyrosine kinase domain-containing kinase (SNK-5). "The activation of SNK-5 is the precursor to the release of the neurotransmitters in the CNS," explains Wohlman. "That could explain why the effects of these medications may be mediated as much by the release of neurotransmitters as by the receptor, including SERT and BDNF." The research was supported by the National Institutes of Health, the U.S. Department of Education, the Eunice Kennedy Shriver National Institute of Child Health and Human Development, the National Institute of Mental Health, the National Institute on Drug Abuse, the U.S. Air Force Research Laboratory, and the U.S. Department of Defense. Article Link: Kirk T. Oleson, et al. (2012, May): Mechanism-Based Pharmacology of SARMs http://www.sciencedirect.com/science/article/pii/S0098819X12000834 Similar articles: